DAS TIERAUGE English

Chronic Glaucoma in Dogs: Relationships Between Histologic Lesions and the Gonioscopic Diagnosis of Pectinate Ligament Dysplasia.

18. Oktober 2016

Saskatoon – mechentel news – Pectinate ligament dysplasia (PLD) is a common cause of canine glaucoma and the definitive clinical diagnosis is based on gonioscopy. Although the histologic lesions of PLD have been described, it has not been determined whether these changes are specific for PLD or if similar histologicchanges can develop as a consequence of secondary glaucoma. The filtration angles of 61 enucleated canine globes with chronic glaucoma were

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Feline Epitheliotropic Mastocytic Conjunctivitis in 15 Cats.

18. Oktober 2016

Berkeley – mechentel news – Mast cell infiltration occurs in malignant, inflammatory (eg, allergic, infectious), and idiopathic disease processes in humans and animals. Here, we describe the clinical and histological features of a unique proliferative conjunctivitis occurring in 15 cats. Ocular specimens were examined histologically, and polymerase chain reaction (PCR) for feline herpesvirus 1 (FHV-1) was performed on ocular tissues obtained

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Superficial Keratectomy and Conjunctival Advancement Hood Flap (SKCAHF) for the Management of BullousKeratopathy:  Validation in Dogs With Spontaneous Disease.

18. Oktober 2016

Rocklin – mechentel news -To evaluate the efficacy of superficial keratectomy and conjunctival advancement hood flap (SKCAHF) for the treatment of bullouskeratopathy in canine patients., 9  dogs (12 eyes) diagnosed with progressive corneal edema underwent superficial keratectomy followed by placement ofconjunctival advancement hood flaps. The canine patients were examined pre- and postoperatively using in vivo confocal microscopy, ultrasonic pachymetry (USP), and Fourier-domain optical coherence tomography (FD-OCT). All owners responded to a survey

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Selenium Protects Retinal Cells from Cisplatin-Induced Alterations in Carbohydrate Residues.

18. Oktober 2016

Balıkesir – mechentel news – To Investigate alterations in the expression and localization of carbohydrate units in rat retinal cells exposed to cisplatin toxicity, we evaluated putative protective effects of selenium on retinal cells subjected to cisplatin. . Eighteen healthy Wistar rats were divided into three equal groups: 1. Control, 2. Cisplatin and 3. Cisplatin+selenium groups. After anesthesia, the right eye of each rat was enucleated. Histochemically, retinal cells of control

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